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Because it is rapidly metabolized by the liver, has fewer and less intense corticosteroid- type but has not been demonstrated to be an effective drug for long- term therapy of Crohn’s disease.
This book provides information for health professionals who help both pediatric and adult patients navigate through the lifelong shadow of a chronic, probably genetically determined ordeal.
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At the present time, its favorable profile may warrant an initial trial of in children and adolescents requiring steroids with appropriately located mild to moderately active Crohn’s disease. 7. Rosenbach Y, Zahavi I,
Because of the high rate of produced by conventional glucocorticoids as non-systemic glucocorticoid has been introduced in the treatment of is a topically active glucocorticoid with a high
Low bioavailability steroids : Main characteristics Low bioavailability steroids , also called topical steroids , were designed to avoid classical steroid – . Their main features include ( Table III ) : – very high affinity for the
Table 7 Switch from conventional steroids to (9mg) in active and inactive Week O 3 5 7 9 (percentage of patients) 65 55 51 48 43 (total number) 269 191 145 113 90 Treatment
In an attempt to limit the unintended systemic effects of glucocorticoid therapy, novel glucocorticoids have been developed. suppression and treatment for maintaining remission was reported in a pooled analysis of four studies. Results glucocorticoids are effective for the shortterm control of of Crohn’s disease, but they are neither effective nor safe for long-term
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Table 22-4 Uses, Dosage, and for Nonantibiotic Therapeutic Agents Used for Chronic Diarrhea in Cats heart failure 1 mg PO q24h Can lead to some systemic Leukeran
Breeding for resistance and yield in to extract. hid. J. Exp. Biol. 18: 429-432. Chahal
Grain loss due to in bajra hybrids. Indian Phytopathol. 27:254-257. Nene, Y.L. and Singh, S.D. 1976. Downy mildew and of from grain or seed lots of bajra
Tapsoba H and Wilson J P 1996 Pathogenic variation in Puccinia substriata var. indica in the southeastern United States and screening for resistance in germplasm. Plant Dis. 80, 395-397. Thakur D P 1984 Ergot disease of pearl
Epidemiology and control of ergot disease of pearl millet. Seed Sci. Technol. 11:797–806. Thakur DP. 1984. Ergot disease of pearl millet. Rev. Trop. Pl. Pathol. 1:297–328. Thakur RP. 1987. Diseases of pearl millet and their management. pp
Plant Disease Reporter 62:157–160. Thakur, R.P., and S.B. King. 1988. Ergot disease of pearl millet. ICRISAT [International Crops Research Institute for the Semi-Arid Tropics], Patancheru, Andhra Pradesh, India, Information Bulletin No. 24.
Conclusions Soon after the introduction of cms-based singlecross hybrids in 1965, began to cause significant economic losses in India. With a few reports of heavy ergot incidence, especially in hybrid cultivars,
Ergot disease of pearl millet. Review of Tropical Plant Pathology 1:297328. Thakur, D.P. Dang, J.K. (1985). Breeding for disease resistance in pearl millet. Presented at the All India Coordinated Millets Improvement Project Workshop, 26-29
: Toxicological Significance and a Mechanism for Disease Escape P . G . Mantle1 Abstract , caused by Claviceps fusiformis , not only causes crop losses but potentially adds a toxic
Sivaprakasham , K . “ A Note on the Control of . ” Andhra Agric . J . 18 ( 1973 ) : 213 . 41 . Brar , G . S . , Chand , J . N . , and Thakur , D . P . “ Fungicidal Control of Ergot of Bajra . ” Haryana Agric . Univ . J . Res .
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